Ukuvimbela umlenze wesokudla wegoli lepencil

Ukuvimbela umlenze wesokudla wesikhwama se-Hyis - ukuphazamiseka kokuqhutshwa kwe-conduction ngaphakathi kwe-ventricles yenhliziyo. Kwenzeka ku-0.2% yabantu abasha. Njengoba uneminyaka yobudala, ukuvama kwalokhu kwesigamu kunyuke ku-0.45%. Lesi sifo cishe ngaso sonke isikhathi sivela engxenyeni yabantu besilisa.

Isici esiyinhloko esenza ukuthi ukuthuthukiswa kwalesi sifo kuhlaselwe isifo senhliziyo esigabeni esiyingozi. Kubantu kuze kube yiminyaka engamashumi amane, ukuvinjelwa kwesigunxele kwesigxobo akuyona into enobungozi, akunaxhumano ne-pathologies yezincubu zenhliziyo. Kwezinye izimo, ukuthuthukiswa kwalolu hlobo lwe-arrhythmia kunomthelela ekufweni komfutho wegazi ophezulu (60% kuwo wonke amacala). Ezimweni ezikude, kubangelwa ukungabikho komzimba okungezansi (ukuthuthukiswa kwezingxenye zokuqala ze-myocardium). Futhi, ukubukeka kwalesi simo kuthinta ubukhona bezinkinga zenhliziyo: iziphambeko ze-interventricular, septa ye-interrial, ukunciphisa komlomo we-artery yamaphaphu. I-thrombembolia yalesi sitsha, enezifo eziphazamisayo zezifo zomsindo ongapheli, iholela ekusweleni kwe-ventricular efanele, lokhu kungabangela ukuguqulwa komzimba.

Izinto ezibangela ekuthuthukisweni kwalesi simo yi-cardiomyopathies, ukulimala okufubeni kwesifuba, i- dystrophies yama-muscular progression , i- hyperkalemia, ukudlula izidakamizwa (i-Novocainamide, i-quinidine, i-glycosides yemvelo), imisebenzi yokuhlinza kulesi sitho (ventriculotomy eqondile), i-neoplasm yenhliziyo, izinsini zesifo se-syphilitic. Ukuvinjwa okuphelele komlenze wesokudla wesikhwama saKhe kunempawu zokwelashwa zezifo ezibangele. Kwezinye iziguli, izinguquko zenziwa ngesikhathi sokuqhuma: ukuhlukanisa okokuqala (kwenzeka kancane kancane) kanye nezwi lesibili (livela kaningi kakhulu).

Izindlela ezilandelayo zokuxilonga zitholakala ku-electrocardiogram: ukwanda kwinkimbinkimbi ye-ventricular, izinyo eliphezulu R, elibhekwa emtholampilo wokuqala we-thoracic, i-serrated R, futhi izinyo likhonjiswe emkhombeni wesobunxele, onobuningi obuhlukile; Isiqondiso esingavumelani S, ST.

Ukuvimbela umlenze wesokudla wesikhwama saKhe, ukwelashwa okungathi, njengoba kungase kubonakale, kufanele kube sína, akudingi ukusetshenziswa kwamalungiselelo akhethekile. Futhi akudingi ukuvuselelwa kwe-edokardialnoy, okuyizimo ezijwayelekile ezenziwa ngenhloso yokuvimbela uma kwenzeka i-infarction. Uma kunesidingo, ukwelashwa kwezifo okuholela njalo ekubukeni kwaloluhlobo lwe-arrhythmia. Ukuvinjelwa kwesikhwama esifanele sesikhwama, esenzeka kubantu benkampani encane, abangenayo izilonda eziphilayo zezicubu zenhliziyo, kunezibalo ezihle kakhulu. Ukubukeka okukude kwalesi sigamu ngeke kufike ekuvinjisweni okuphelele kwe-AB kwe-AB node.

Ngokusho kocwaningo, okwenzeka ngesikhathi esisodwa kulezi zifo kubangela ukuphazanyiswa okuncane kokuqhutshwa kwe-atria kuya kwi-ventricles. Kodwa-ke, kungenzeka ukuthi inkambo yezokwelapha yokuvimbela lesi sifo ibuhlungu njengoba ukuqhutshwa komfutho emkhakheni we-AB kunciphisa. Uma lolu hlobo lwe-arrhythmia luthinteka ngokuqondile nesifo somfutho wegazi, inkambo yalesi sifo iyinkimbinkimbi kakhulu, ukulimala ngaphansi kwezimo ezinjalo kuyaba phezulu. Isibonakaliso esingathandekiyo kuba khona kokuphelelwa yisifo senhliziyo, ukuhluleka kwenhliziyo, ukweluleka kwesikhathi se-systolic. Ukuvimbela umlenze wesokudla we-Hisb, okwenzeka ngokumelene nesizinda se- infarction ye-myocardial, cishe awusoze wadlula kunoma yikuphi ukuhleleka okuhlobene nokuphulwa komsebenzi ku-node ye-AB. Ngakho-ke, lolu hlobo lwe-pathology cishe alunalo izinga eliphezulu lokufa. Kodwa-ke, phambi kokucindezeleka okukhulu kwenhliziyo, kungenza kube nzima nakakhulu ukwelashwa, okungase kuqiniseke ukuthi akusebenzi.